2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

We quantified UBXN2A degrees in Panels 5A (cytoplasmic Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cellsread more...

2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

To additional understand the pre-apoptotic Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis read more...

2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

Since UBXN2A functions as a Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells functional inhibito read more...

2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

Posttranslational modification such as phosphorylation, ubiquitination, and sumoylation has been Etoposide shown to control nuclear export of various proteins by read more...

2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

Our final results confirmed that UVB irradiation of HCT-116 cells brought about an Etoposide increase in UBXN2A degrees in the cytoplasm in a dose-dependent metho read more...

2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

HCT-116 and LoVo cells were being purchased from ATCC (American Etoposide Form Lifestyle Collection) and had been developed in suitable media, supplemented with < read more...

2 years ago

Nucleocytoplasmic Translocation of UBXN2A Is Required for Apoptosis during DNA Damage Stresses in Colon Cancer Cells

The subcellular localization, expression amount, and action of anti-most cancers proteins change in Etoposide response to intrinsic and extrinsic mobile stresses read more...